11 hours ago The patient had an emergency exploratory surgery this morning for a suspected perforated viscus. The patient had an exploratory laparotomy, repair of colon, diverting colostomy. Ascites fluid was felt to be infected. ID evaluation was requested. The patient is currently in the recovery department. She says she is in a lot of abdominal pain. >> Go To The Portal
The guidelines recommend a diagnosis of peritonitis with at least two of the following: Clinical signs and symptoms such as abdominal pain and/or cloudy effluent Dialysis effluent white cell count >100 cells/µL after a dwell time of ≥2 hours and >50% of cells are polymorphonuclear Positive dialysis effluent culture
The PD effluent should be tested for cell count, differential, Gram stain, and culture when peritonitis is suspected. Patients with peritonitis usually present with cloudy effluent and abdominal pain.
Peritonitis is the inflammation of the peritoneum, the serous membrane lining the abdominal cavity and covering the viscera. Usually, it is a result of bacterial infection; the organisms come from diseases of the GI tract, or in women, from the internal reproductive organs.
• Abdominocentesis is the preferred diagnostic method for confirming peritonitis. • When abdominal fluid cytology reveals degenerative neutrophils and intracellular bacteria, confirming a diagnosis of septic peritonitis, emergency surgical exploration of the abdomen is indicated.
Peritonitis is the inflammation of the peritoneum, the serous membrane lining the abdominal cavity and covering the viscera.
Inflammation. An inflammation that extends from an organ outside the peritoneal area such as the kidne ys could cause peritonitis. Bacteria. The most common bacteria implicated are Escherichia coli, Klebsiella, Proteus, Pseudomonas, and Streptococcus.
Oxygen therapy. Oxygen therapy by nasal cannula or mask generally promotes adequate oxygenation. Antibiotic therapy. Antibiotic therapy is initiated early in the treatment of peritonitis.
Response. The immediate response of the intestinal tract is hypermotility, soon followed by paralytic ileus with an accumulation of air and fluid in the bowel.
The pathophysiology of peritonitis involves: Leakage. Peritonitis is caused by leakage of contents from abdominal organs into the abdominal cavity. Proliferation. Bacterial proliferation occurs. Edema. Edema of the tissues occurs, and exudation of fluid develops in a short time. Invasion.
Sepsis. Sepsis is the major cause of death from peritonitis. Shock. Shock may result from septicemia or hypovolemia. Intestinal obstruction. The inflammatory process may cause intestinal obstruction, primarily from the development of bowel adhesions.
The nurse should educate the patient and the family about the care for incisions and drains if the patient will be sent home with the drains still in place. Referral. Referral for home care may be indicated for further monitoring and patient and family teaching.
When bacteria invade the peritoneum due to an inflammation or perforation of the GI tract peritonitis usually occur s. Bacterial invasion usually results from appendicitis, diverticulitis, peptic ulcer, ulcerative colitis, volvulus, abdominal neoplasms, or a stab wound. It may also be associated with peritoneal dialysis.
Peritonitis is the acute or chronic inflammation of the peritoneum, the membrane that lines the abdominal cavity and covers the visceral organs. Inflammation may extend throughout the peritoneum or may be localized as an abscess. Peritonitis commonly decreases intestinal motility and causes intestinal distention ...
Administer antimicrobials: gentamicin (Garamycin), amikacin (Amikin), clindamycin (Cleocin), via IV/peritoneal lavage.
Oliguria develops as a result of decreased renal perfusion, circulating toxins, effects of antibiotics. Maintain strict aseptic technique in care of abdominal drains, incisions and/or open wounds, dressings, and invasive sites. Cleanse with appropriate solution.
Note changes in mental status: confusion, stupor, altered LOC. Hypoxemia, hypotension, and acidosis can cause deteriorating mental status.
mortality is 10% with death usually a result of bowel obstruction. The peritoneum is sterile, despite the GI tract normally contains bacteria.
Matt Vera, BSN, R.N. Matt Vera is a registered nurse with a bachelor of science in nursing since 2009 and is currently working as a full-time writer and editor for Nurseslabs. During his time as a student, he knows how frustrating it is to cram on difficult nursing topics. Finding help online is nearly impossible.
Introduction to Peritonitis in Peritoneal Dialysis. Peritonitis is the inflammation of the peritoneum or peritoneal membrane. This type of inflammation typically has an infectious etiology that is mainly caused by bacteria (~80% of cases). Bacterial infections mainly come from contamination during a peritoneal dialysis (PD) session.
In order to prevent peritonitis, both exit-site infections and catheter-tunnel infections must be prevented. The guidelines recommend prophylactic antibiotic therapy right before insertion of the catheter. No single catheter has been shown to have better outcomes than another in regards to peritonitis rates but ISPD does recommend disconnect systems with a “flush before fill” design with continuous ambulatory PD (CAPD). In addition, the guidelines recommend training programs for patients and that they be conducted by health care providers with experience. To prevent exit-sit infections, the guidelines recommend topical antibiotics like mupirocin or gentamicin daily. If an exit-site infection develops, treatment should be prompt and targeted to decrease the risk of peritonitis development.
Peritonitis is a major complication of PD due to the structural changes in the peritoneal membrane that result from PD. Approximately 4% of peritonitis episodes result in death. Since peritonitis is one of the more common complications of PD, it has caused patients to switch from this modality to hemodialysis.
Cloudy effluent usually represents infectious peritonitis, but there could be differential diagnoses aside from infectious peritonitis, such as chemical peritonitis which is a possible side effect of calcium channel blockers, eosinophilia of the effluent, hemoperitoneum, malignancy, chylous effluent, or specimen taken from a dry abdomen.
The guidelines recommend repeat WBC count with differential if the effluent yields no growth after 3 days. If the peritonitis has not clinically improved at day 3, special culture techniques that test for mycobacteria, nocardia, legionella, filamentous fungus, or other fastidious bacteria should be considered.
Catheters should be removed immediately once fungi are identified for improved outcomes and reductions in mortality.
Most PD patients with peritonitis will show considerable clinical improvement within 48 hours of initiating therapy.